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A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

Obese women with PCOS have lower vitamin D levels.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

The vitamin D receptor can work without its usual activating molecule.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

Vitamin D3 applied to mouse skin caused more wrinkles and sagging due to changes in the skin's outer layer.

Vitamin D3 affects cell differentiation in specific skin areas.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Topical tacalcitol can treat psoriasis by acting directly on skin cells.

Estrogen deficiency can reduce the enzyme activity needed to activate vitamin D.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

A vitamin D receptor mutation causes rickets and affects immune responses.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Vitamin D3 may prevent hair loss from chemotherapy, but side effects and cancer cell protection are concerns.

Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

A girl had rickets due to a gene mutation affecting vitamin D response.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Vitamin D affects skin and hair by influencing cell behavior and melanin production.

Vitamin D is crucial for bone health and preventing serious diseases.

Rubbing vitamin D3 on skin can help prevent hair loss from chemotherapy and slow breast tumor growth in mice.

Vitamin D receptor is crucial for skin wound healing.