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Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

Skin biopsy is crucial for diagnosing unknown baldness causes.

Activating β-catenin can turn skin cells into hair follicles.

The hair follicle infundibulum plays a key role in skin health and disease, and understanding it better could lead to new skin disease treatments.

Sgk3 is essential for normal hair follicle growth and maintenance.

N-WASP is essential for normal hair growth in mice.

The Gsdma3 gene is essential for normal hair development in mice.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Claudin 6 is crucial for normal skin and hair development.

The protein aPKCλ is crucial for keeping hair follicle stem cells inactive and for hair growth and regeneration.

Cathepsin L is essential for normal hair growth and development.

Dlx3 is essential for hair growth and regeneration.

Androgens like testosterone affect hair growth and oil production differently across body parts and individuals.

Akt2 and SGK3 are both important for normal hair growth and development.

BMP signaling controls hair growth and skin color.

Hair growth and development are controlled by specific signaling pathways.

EGFR is essential for normal hair development and follicle differentiation.

The study found that a specific area of the hair follicle helps start hair growth by reducing the blocking effects on certain cells and controlling growth signals.

Smad4 is important for healthy hair follicles because it helps produce a protein needed for hair to stick together and grow.

Leptin helps start the growth phase of hair.

aPKCλ is crucial for keeping hair follicle stem cells inactive and maintaining normal hair growth.

Hairless protein reduces Msx2 gene activity, affecting hair follicle development.

Disrupted stem cell signals in hairpoor mice cause hair loss.

Basement membrane changes are crucial for hair follicle development.

aPKCλ is essential for hair follicle stem cell maintenance and wound healing.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

Oestrogen and thyrotropin-releasing hormone affect prolactin and its receptor in human skin and hair, suggesting new treatment options for related conditions.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Removing Gata6 causes hair follicle and sebaceous duct enlargement.