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Alopecia areata may be caused by antibodies targeting specific hair follicle proteins, hindering hair growth.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Topical immunotherapy for alopecia areata may work by creating immune cell clusters in the skin.

The CD44-CD49d complex boosts T cell activation and survival in autoimmune disease.

Arg1+ macrophages may play a role in Alopecia Areata, offering new treatment targets.

Autoimmune reactions may cause both alopecia areata and HAM.

HLA-DRB5 and other genes may be linked to alopecia universalis.

Alopecia areata involves unique activation of certain immune cells.

Alopecia areata is linked to immune system differences, with specific biomarkers like CXCL9 and CXCL10 being key for diagnosis and potential treatment targets.

Alopecia areata involves specific T-cells, unlike androgenetic alopecia.

Certain interactions help prepare the androgen receptor for pairing and activation, which is important for its role in development and disease.

Pertussis toxin may contribute to hair loss in alopecia areata.

Certain genes are linked to the risk of developing Alopecia Areata.

Alopecia areata involves specific immune cells, offering potential treatment targets.

The rs231775 genetic variant is linked to a higher risk and severity of Alopecia Areata in males.

ASLAN004 was safe and well-tolerated, supporting further development for treating certain diseases.

Certain genetic variants in the androgen receptor are linked to higher PSA levels, potentially affecting prostate cancer screening outcomes.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

The Fas/FasL pathway may play a role in alopecia areata.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Antibody drug conjugates show promise in targeting cancer cells while reducing harm to healthy cells.

Amotosalen-treated donor T-cells can prevent late CMV infection after bone marrow transplants.

The ALADIN score can predict how well patients with alopecia areata will respond to JAK inhibitor treatments.

Alopecia areata is an autoimmune disorder causing hair loss, linked to specific hair follicle antigens and genetic factors.

Targeting CXCL10 may help treat alopecia areata.

The role of γδT-cells in causing alopecia areata remains unclear.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.