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A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Removing anthrax toxin receptor 1 in pigs prevents Senecavirus A infection and causes a rare disease similar to GAPO syndrome.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

The document concludes that certain rats and mice are useful for studying hair loss in humans and testing treatments.

MOF controls skin development by regulating genes for mitochondria and cilia.

Overexpressing COX-2 in mice skin reduces skin tumor development.

Adult mice with CBS deficiency show minimal health issues and normal lifespan despite high homocysteine levels.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

AIRE deficiency causes hair loss similar to alopecia areata in mice.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

Mouse models help study genetic skin diseases.

Removing the ATR gene in adult mice causes rapid aging and stem cell loss.

Iron deficiency in mothers causes hair loss in their baby mice.

Apremilast may help treat hair loss in alopecia areata.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

A new mutation in mice causes crooked whiskers and messy hair.