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Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Social isolation makes mice more sensitive to alcohol's effects on brain function.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

The angora mouse mutation causes long hair and hair defects due to a gene deletion.

The enzyme sEH is important for hair growth and its inhibition could help treat hair loss.

Hedgehog signaling is essential for normal sebaceous gland development and affects keratin 6a expression.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Whiskers can form without sensory nerves or Foxd1, thanks to Meis2 in mesenchymal cells.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Increasing SSAT makes skin more prone to cancer.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

Mouse models help target specific genes in lymphatic cells for research.

The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.

Diabetic neuropathy in mice is linked to poor mitochondria function and lower brain hormone production.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Multiple mouse desmoglein 1 isoforms have distinct roles in skin and hair development.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

The hairless mutant gene causes early hair loss and affects skin and thymus development in mice.

Blocking hair follicle development stops key gene signals needed for hair growth in mice.

African spiny mice can regenerate skin and hair after wounds due to specific tissue mechanics.

The mouse model could be useful for baldness research and testing treatments like testosterone, cyproterone acetate, and minoxidil.

Blocking TGFβ-RI signaling enhances surface ectoderm differentiation from human stem cells.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

A new rat strain with a specific gene mutation causes hair loss and kidney issues.

Ovariectomized mice mimic postmenopausal hair loss, and estradiol helps maintain hair density.