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The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Matriptase imbalance contributes to cancer development and spread.

ROR2 is essential for hair follicle stem cell renewal and maintenance.

AP-1 controls tumor cell type by affecting key signaling pathways.

β‐thymosins, especially thymosin β4, help in heart protection, wound healing, and hair growth by stabilizing actin in cells.

Imaging living plant cells shows dynamic changes in actin and endoplasmic reticulum linked to root hair growth.

CTIP2 may help in skin development and maintenance.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Sfrp2 increases during hair follicle catagen phase and slows keratinocyte growth.

Blocking mTORC1 reduces skin tumor growth in mice.

Ets2 gene is crucial for placental development in mice.

E-cadherin is important for cell movement in electric fields, and the new tracking method works well.

Akt2 and SGK3 are both important for normal hair growth and development.

All-trans retinoic acid slows cell growth and increases cell death in goat hair follicles through a specific pathway.

Ku80 is a key receptor for Thymosin β4, affecting cell migration and wound healing.

Lower MC2R expression may contribute to alopecia areata.

Targeting the actin cytoskeleton could improve skin healing and reduce scarring.

Activin A and Follistatin affect how mouse hair follicles grow.

Thymosin β4 helps with healing, inflammation, and organ protection.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

Celsr1 is crucial for skin cell alignment, while Celsr2 has little effect on this process.

RORA affects hair follicle stem cells' structure and movement, potentially helping treat hair loss.

Raptor and Rictor have stable expression in hair cycles, with Raptor marking stem cells and Rictor involved in hair shaft formation.

KRT2.13 gene is similar to KRT2.9 but not expressed in hair follicles.

The enzyme Dgat1 is essential for healthy hair and skin by controlling retinoid levels.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting connective tissue sheath contraction may improve hair growth in male pattern baldness.