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C57BL/6 mice and SD rats have different sweat gland and hair follicle patterns, useful for skin research.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

Too much male hormone in mothers can negatively affect the sexual behavior of both male and female baby mice.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Key genes linked to hair growth and cancer were identified in hairless mice.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Acid inside cells speeds up aging and turns on aging signs in mice.

Pure carbon nanotubes are safe for mice, but impure ones cause immune issues and hair loss.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Copper injections improved symptoms and prevented brain damage in brindled mice.

The mouse models are effective for testing new hair loss treatments.

The role of γδT-cells in causing alopecia areata remains unclear.

Activating Tgr5 may help treat hair loss and bone loss.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Artificially inducing hair regrowth in mice can change the normal pattern and timing of hair growth, with minimal color differences between old and new fur.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

The method helps study hair follicle stem cells and calcium signals in mouse skin.

The Gsdma3 gene is essential for normal hair development in mice.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

Meis2 is essential for touch sensation and nerve function in mice.

Thymoma may link myasthenia gravis, nail dystrophy, and alopecia areata.

Fat stem cells from diabetic mice can still help heal wounds.

A new mouse model helps study melanocyte cells using GFP expression.

Stress can slow hair growth and affect skin color by impacting the body's stress response system.