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New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Feline cutaneous lymphocytosis is likely reactive, while canine cutaneous lymphocytosis needs more study to understand its nature.

T cells with memory features grow in number and gather around hair follicles when there are not enough immune cells.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Human thymus has stem cells that can self-renew and maintain their identity.

Alopecia areata is linked to reduced T cell function and auto-immunity.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

CD8+ T cells re-expressing CD45RA may predict treatment resistance in severe alopecia areata.

Chronic refractory alopecia areata has more skin-resident memory T cells, and JAK inhibitors may help reduce them.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

Increased regulatory T cell activity may lead to better outcomes in acute diffuse and total alopecia.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Increased Treg cells and IL-10 may help quick recovery in acute diffuse and total alopecia.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

ILC1-like cells may contribute to hair loss in alopecia areata.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Special cells can help regrow hair in alopecia areata.

Poor response to topical immunotherapy in alopecia areata patients is linked to impaired cell responses.

Alopecia areata involves specific immune cells, offering potential treatment targets.

Different γδ T cell types have unique roles in causing alopecia areata.

Enhancing Tregs can protect against alopecia areata.

CD4 T cells need IFN-γ to cause hair loss in alopecia areata.

γδTregs may help treat autoimmune diseases like alopecia areata by promoting hair regrowth and reducing immune attacks.

Targeting specific T cells may help treat alopecia areata.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

A specific group of immune and skin cells may cause chronic inflammation in atopic dermatitis.