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Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Peptide nanoparticles can effectively deliver CRISPR-Cas9 to target KRAS mutations in cancer.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

The E2 protein affects gene activity in hair follicles of mice.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

A new gene, mrp4, is found in mice and may play a unique role in hair follicle development in tails and ears.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

Transplanted bone marrow cells actively move, form clusters, and grow after transplantation.

Scientists made a mouse that can be made to lose hair and then grow it back.

A gene mutation in mice causes permanent hair loss and skin issues.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

Transgenic sheep cells with spider silk gene were successfully created for future sheep hair expression.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

Overexpressing the glucocorticoid receptor in mice leads to abnormal skin development and reduced inflammation.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Blocking mTORC1 reduces skin tumor growth in mice.

BLMP-1 is important for regular molting and gene expression cycles in worms.

The DiLiCre mouse model is an effective tool for precise genome editing using light.