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A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Mesenchymal MEIS2 is essential for whisker development without needing sensory nerves.

Excess vitamin A causes lasting gland changes in mouse hair follicles.

Mutations in TBX3 cause horses to have more even hair color instead of Dun camouflage.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

The rhg mutation in mice affects the Oat gene, causing hair growth issues and other symptoms.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

Adult mice can grow new hair from skin wounds.

A zinc-deficient diet stunted growth and affected organs in mice, with C57BL/6J mice showing more severe symptoms.

Tortoiseshell tomcats with XX/XY chimerism can be fertile.

Mouse hair follicles age, causing more white hairs due to fewer pigment stem cells.

Newborn mice need colostrum for zinc to grow normally.

Two genes, Tabby and Ticked, determine cat coat patterns.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Two mouse mutations cause similar hair loss despite different skin changes.

BMI1 is essential for preventing hair greying and maintaining hair color.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

The mouse model could be useful for baldness research and testing treatments like testosterone, cyproterone acetate, and minoxidil.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Mouse spermatogenesis shows that stem cells can behave flexibly and move widely in open environments.