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Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Mouse nails are similar to human nails, making them useful for studying nail diseases.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

The mouse model could be useful for baldness research and testing treatments like testosterone, cyproterone acetate, and minoxidil.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

A testosterone-induced hair loss model in mice was successfully created for future research and treatment testing.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Neurosteroids help reduce seizures, but their withdrawal increases seizure activity.

The treatments stopped hair regrowth in mice.

The complex oriental medicine extract was as effective as minoxidil for hair regrowth in mice without adverse effects.

The improved genome of the African spiny mouse helps study its tissue regeneration.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

A new mouse model effectively mimics vitiligo for research and drug testing.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Scientists made a mouse that can be made to lose hair and then grow it back.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Mouse models help study genetic skin diseases.

The CUBIC protocol allows detailed 3D visualization of proteins in mouse skin biopsies.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.