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Mitochondrial Complex I reduces inflammation and increases bone breakdown by affecting certain immune cells.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

Cortisol increases a specific protein in bone cells, which may help explain its negative effects on bone.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Suppressing eIF4E can prevent hair loss from chemotherapy.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

Changing the diet of mice lacking the enzyme CBS can affect symptoms related to the genetic condition.

Suppressing ODC activity reduces tumor growth in hair follicles.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Disrupting Bcl-xL in mice reduces skin cancer risk.

Blocking CYP17A1 enzyme may help improve certain brain function issues related to dopamine.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Polycomb Repressive Complex 1 is crucial for keeping stem cells stable and maintaining healthy adult tissues.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.

RSPO1 could help create new diabetes treatments by increasing pancreatic β cells.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

CS12192 effectively treats alopecia areata with better safety than current options.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D and glucocorticoids affect bone growth, metabolism, and immune response.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

Targeting Sirt1 can reduce androgen levels and slow glioblastoma growth.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

A specific genetic change is linked to mental disorders, intellectual disability, and possibly autoimmune disease in a family.

The study found two new mutations in a Chinese patient with severe biotinidase deficiency.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Disrupted citric acid metabolism stops hair growth.