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A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

Blocking the CCR5 receptor may be a new way to treat hair loss from alopecia areata.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

FoxN1 gene is essential for proper thymus structure and preventing hair loss.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Deleting MPZL3 increases skin oil production and reduces body fat.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

Phospholipase Cδ1 is crucial for normal skin and hair development.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Progesterone's seizure-reducing effects are mainly due to allopregnanolone, not progesterone receptors.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

QLT0267 stops hair follicle cell growth and movement.

Quercitrin may help treat Alzheimer's by boosting brain cell signaling without causing tumors.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Blocking CCR5 can prevent and improve hair loss in alopecia areata.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Disrupted citric acid metabolism stops hair growth.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

Injury boosts normal skin cell growth, reducing cancer cell advantage.

AD198 is more effective than doxorubicin in stopping certain dog cancer cells.

MFN2 mutations cause mitochondrial problems, leading to more upper body fat and lower leptin levels.

β-catenin, Shh, and Bmp signaling control hair follicle development.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Overexpressing noggin in mice causes severe osteoporosis.

Alkaline Ceramidase 1 prevents early hair loss in mice by keeping hair follicle stem cells balanced.

Lack of vitamin D receptor causes hair loss in mice by allowing certain genes to overactivate.

Deleting Rac1 in the skin depletes stem cells and damages hair follicles.