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Activating cannabinoid receptor 1 may help manage psoriasis by reducing certain keratins.

Skin tumors with CYLD cutaneous syndrome show more NF-κB activity and less organized collagen.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

Targeting ornithine decarboxylase can help prevent skin cancer.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

New therapies and trials are needed for Merkel cell carcinoma, a tough skin cancer.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Dabrafenib can cause skin growths and sometimes low-grade skin cancer.

LRIG1 is linked to better survival in Merkel cell carcinoma.

Rabies virus was found in specific skin cells of rabid dogs' muzzles, suggesting these cells could help diagnose rabies.

Inhibiting ornithine decarboxylase may help prevent certain skin cancers.

A mycobacterial protein shares a similar region with a human skin protein, possibly affecting skin diseases.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

PDCD4 is important for controlling skin cell growth and healing.

Keratinocytes can reduce the survival of certain melanoma cells, suggesting new therapy paths.

MC-1R in skin cells may influence inflammation and collagen production.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

K31 can identify clear secretory cells in human sweat glands.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

CD98hc's role in skin health decreases with age.

High MUC-18/MCAM levels in blood indicate a worse outlook for melanoma patients.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

TCDD disrupts skin stem cells, causing skin issues like chloracne.

Kremen is crucial for proper development and preventing tumors by regulating Wnt signaling.

REV7 is crucial for genome stability and cancer treatment, making it a potential target for therapy.