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People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Some patients with a type of skin lymphoma can experience a rare, non-scarring hair loss that looks like another hair loss condition but has distinct features.

The growth of the Epstein-Barr virus in the patient's cells was linked to the worsening of her lymphoma.

FOXN1 gene variants cause low T cells and immune issues from birth.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Androgens may influence T cells, contributing to higher autoimmune liver disease risk in women.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Psoriasis patients' immune response to a hair protein depends on their specific gene type.

IL-17-producing γδ T cells help improve bone healing.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Targeting CD200 could be a new treatment for rheumatoid arthritis.

New imaging technology can show up to 40 different markers in hair loss tissue, helping to understand hair disease better.

γδ T cells can prevent and treat alopecia areata, offering a new therapy option.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.

Autoimmune hair loss may be linked to increased Th1 and Th17 cells and decreased Th2 cells.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

Clonally expanded CD8+ T cells cause alopecia areata.

Dexamethasone-primed stem cell media shows promise in treating lupus by reducing symptoms and inflammation.

A new mouse model effectively mimics vitiligo for research and drug testing.

Early intervention with JAK inhibitors may prevent alopecia areata progression.

γδ T cells are essential for wound healing after poxvirus infection.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Understanding T cells and signaling pathways can lead to better treatments for hair loss.

IFN-γ production by CD4 T cells is crucial for causing alopecia areata.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

CD44 variant changes start alopecia areata, but don't maintain it.