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Tofacitinib and low-dose IL-2 may help maintain hair regrowth in alopecia areata without ongoing treatment.

Using CD123 to detect certain immune cells helps diagnose a type of hair loss condition.

CD28 is a promising target for treating alopecia areata with belatacept.

CD28 is a promising target for treating alopecia areata with belatacept.

Ritlecitinib may help treat alopecia areata by protecting hair follicles.

IL-17-producing γδ T cells help improve bone healing.

Interferon, especially alfa interferon, is an effective treatment for cutaneous T-cell lymphoma with manageable side effects.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Intestinal intraepithelial lymphocytes are crucial for gut immunity and maintaining the mucosal barrier.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

OCT4B1 and SOX-2 levels are higher in Crohn's and ulcerative colitis patients, possibly aiding tissue repair.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

γδ T cells are essential for wound healing after poxvirus infection.

Blocking certain proteins on immune cells may help treat alopecia areata.

Mast cells and CD8 T cells interact closely in skin diseases, affecting each other's behavior and contributing to conditions like psoriasis and eczema.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

Tislelizumab can cause cutaneous lupus erythematosus.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

CD2 might be a new treatment target for patchy alopecia areata.

Blocking interferon-gamma might help treat various autoimmune diseases.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

IL-25 helps heal diabetic wounds by improving blood vessel and skin cell functions.

Anti-CD19 therapy may help treat SLE and NMOSD.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

New therapies like JAK inhibitors and stem cells show promise in treating vitiligo.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Controlling Tslp can improve health in AEC syndrome patients.