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Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The research identified new skin traits in mice, some linked to human skin conditions.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Iron deficiency causes hair loss by affecting hair differentiation and cycling.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Disrupting Bcl-xL in mice reduces skin cancer risk.

The hairless mutant gene causes early hair loss and affects skin and thymus development in mice.

DKK4 can be used to improve wool quality in Zhexi Angora rabbits.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

CD34 is crucial for skin tumor development in mice.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

A new mouse model helps study melanocyte cells using GFP expression.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

KLF4 is important for maintaining skin stem cells and helps heal wounds.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.