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Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.

CCL5 is important for the hair growth potential of human dermal papilla cells.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Tislelizumab can cause cutaneous lupus erythematosus.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

Researchers created a cell line to study hair growth and found specific genes affected by dihydrotestosterone.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

Donor lymphocyte infusions effectively treated leukemia relapse but caused vitiligo and alopecia areata.

Hair follicle cells resist turning into skin cells.

Keratin 15 cells from hair follicles help develop and maintain skin tumors in mice.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

CD27 and IL-35 can help diagnose alopecia areata linked to bacterial infections.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Rat hair follicle stem cells can be successfully cultured and may be useful for creating tissue-engineered hair, vessels, and skin.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

CD1d expression in scalp skin and hair follicles changes with the hair cycle and may help protect against microbes.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

Mononuclear cells may protect against certain chemotherapy-induced hair loss.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Researchers found a good way to isolate hair follicle stem cells from newborn goats for further study.

CD10 helps distinguish between basal cell carcinoma and benign hair follicle tumors.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

BCC can protect heart cells from damage caused by oxidative stress.

Adipose-derived stem cells help heal radiation skin damage by reducing cell death and inflammation.