Search

everythinglearnresearchcommunity

for

Search:↓

Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

Transplanted bone marrow cells actively move, form clusters, and grow after transplantation.

MC-1R in skin cells may influence inflammation and collagen production.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

IL-6 from stem cells helps repair skin and grow hair.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Skin stem cells help create protective immune cells during wound healing.

Blocking IL-12 can help treat alopecia areata by preventing hair follicle immune issues.

Bipotent dermal stem cells and specific macrophages are crucial for hair regrowth and regeneration.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Machine learning can help identify biomarkers for personalized Pemphigus vulgaris treatment.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

CD44 variant changes start alopecia areata, but don't maintain it.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Certain microRNAs found in normal cells can effectively suppress various cancers.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Ng2+ perivascular cells in mouse skin come from specific fibroblast types and help in tissue repair.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.