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RC48 shows promise for treating certain advanced cancers, but more research is needed.

CXCR2 in skin cells promotes tumor growth.

Recognizing RSCC is crucial due to its aggressive nature and high risk of poor outcomes.

Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

A new one-step test can quickly identify skin cancer during surgery.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Blocking certain immune signals can reduce skin damage from radiation therapy.

A mycobacterial protein shares a similar region with a human skin protein, possibly affecting skin diseases.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

CS12192 effectively treats alopecia areata with better safety than current options.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

ALRN-6924 may prevent hair loss caused by chemotherapy.

BerEP4 and CD34 staining can help tell apart tricholemmoma from basal cell carcinoma.

Targeting CD200 could be a new treatment for rheumatoid arthritis.

Targeting Keratin 17 may help overcome cancer therapy resistance.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

Nonimmunogenic forms of keratins K71 and K31 can delay and prevent alopecia areata.

Targeting CD169+ skin macrophages may help treat psoriasis.

CD2 might be a new treatment target for patchy alopecia areata.

Checkpoint inhibitor therapy can cause skin issues, from mild rashes to severe reactions.

Merkel cell carcinoma is most likely to recur within two years of diagnosis, and factors like immune suppression, being over 75, and male sex increase this risk.

ABCG2 protein marks stem-like skin cells in human epidermis.

CD8+ T cells re-expressing CD45RA may predict treatment resistance in severe alopecia areata.

Contact immunotherapy can change immune responses in alopecia areata, suggesting new treatment targets.

Skin cells can naturally limit the growth of cancerous changes by balancing cell renewal and differentiation.

NSC167409 can effectively inhibit the virus causing hand, foot, and mouth disease.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.