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CD28 is a promising target for treating alopecia areata with belatacept.

CD28 is a promising target for treating alopecia areata with belatacept.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

Vesiculobullous lesions should be considered part of canine cutaneous epitheliotropic T-cell lymphoma.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Blocking IL-8 can reduce skin rashes from cancer treatment.

Three characteristics of plasmacytoid dendritic cells help tell apart lupus-related hair loss from LPP.

CD1d expression in scalp skin and hair follicles changes with the hair cycle and may help protect against microbes.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Thymus-derived Tregs, not peripherally-derived Tregs, primarily regulate type 1 diabetes in the NOD mouse model.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Understanding T cells and signaling pathways can lead to better treatments for hair loss.

BST2 protein and certain T cells increase in early alopecia areata.

Targeting Vδ1+T-cells may help treat alopecia areata.

CD34 is crucial for skin tumor development in mice.

Activating TLR9 helps heal large wounds and regrow hair by involving a specific type of immune cell.

CD34 marks potential stem cells in dog hair follicles.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Controlling Tslp can improve health in AEC syndrome patients.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Monoclonal antibody B72.3 selectively reacts with certain dog tissues, mainly in the gastrointestinal and respiratory tracts.

CD10 helps distinguish between basal cell carcinoma and benign hair follicle tumors.

CD34+ hair follicle stem cells can become melanin-producing cells for treating skin conditions.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.