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The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Antibodies help identify glycoproteins in normal skin and tumor cells.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Human thymus has stem cells that can self-renew and maintain their identity.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Microglia are essential for zebrafish brain repair by controlling inflammation.

Regulatory T cells help prevent autoimmunity and have potential for treating autoimmune diseases.

CXXC5 can both suppress and promote cancer, making it a complex target for treatment.

Some brain cancer cells avoid immune system detection, and certain treatments could target this to slow their growth; also, certain fat cell precursors help regenerate hair and skin after injury.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

HOTTIP and miR-10b contribute to glioma therapy resistance by affecting cell behavior, suggesting they could be targets for treatment.

Regulatory T cells adapt to different environments to control inflammation and support tissue repair.

The study suggests a link between varicella-zoster virus and segmental vitiligo, with evidence of the virus disrupting skin pigment cells.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

Entospletinib effectively prevents eye and skin GVHD in mice.

A specific genetic trait in tumor cells is linked to longer survival without disease in certain lymphoma patients.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.