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Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Certain NK cell changes in blood may indicate alopecia areata progression.

Hair follicles help skin immune recovery after UVB exposure.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

Using CD123 to detect certain immune cells helps diagnose a type of hair loss condition.

Tph cells are linked to the severity of systemic lupus erythematosus.

Three characteristics of plasmacytoid dendritic cells help tell apart lupus-related hair loss from LPP.

Blocking certain proteins on immune cells may help treat alopecia areata.

Both induced and spontaneous AA lymphocytes can cause alopecia areata in mice.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Bendamustine combined with tucidinostat may effectively treat adult T-cell leukemia.

IL-18 signaling helps mature Tregs move into the thymus.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

T cell subsets are crucial in kidney cancer, and a new model predicts patient outcomes using key genes.

Poor response to topical immunotherapy in alopecia areata patients is linked to impaired cell responses.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

Increased regulatory T cell activity may lead to better outcomes in acute diffuse and total alopecia.

CD1d expression in scalp skin and hair follicles changes with the hair cycle and may help protect against microbes.

UV light helped human hair transplants survive in mice without broad immunosuppression.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Activating TLR9 helps heal large wounds and regrow hair by involving a specific type of immune cell.

Monoclonal antibody B72.3 selectively reacts with certain dog tissues, mainly in the gastrointestinal and respiratory tracts.

Amotosalen-treated donor T-cells can prevent late CMV infection after bone marrow transplants.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

VDC-1101 shows potential as a treatment for canine cutaneous T-cell lymphoma.