Search

everythinglearnresearchcommunity

for

Search:↓

Hox proteins help maintain keratinocyte identity by regulating miRNA expression.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

ADF8 and ADF11 help root hairs grow by responding to hormones and environmental signals.

The update highlights that non-classic congenital adrenal hyperplasia is common in women with excess male hormones, requires specific hormone tests for diagnosis, and has various treatment options depending on age and symptoms.

MARCKSL1 is important for wound healing and could be a target to reduce scarring.

The research identified and described a gene important for hormone conversion in endangered catfish, which varies in activity during different reproductive stages and after hormone treatment.

The GPRC6A-Duox1 axis helps control hair growth and loss by affecting hydrogen peroxide production.

The new antibody, TYHF-1, specifically targets certain hair-related structures.

The HIF-2α/ARNT complex is important for hair follicle development by controlling cell growth.

Scientists identified a unique type of human skin stem cell that could help with tissue repair.

ALX4 is crucial for normal craniofacial and hair development, with specific roles in different cell types.

Mutations in the SRD5A3 gene cause a new type of glycosylation disorder by blocking the production of a molecule necessary for protein glycosylation.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

CD200 is not a reliable marker for identifying stem cells in all skin types.

The May issue discussed publishing agreements and four studies on cholesterol in hair, cancer cell changes, hormones in the uterus, and skin protein effects.

HS needs personalized treatment plans and more research.

Minoxidil treatment improves heart defects in a DiGeorge syndrome model.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

The research identified specific genes that are active in the cells crucial for hair growth.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Hormonal imbalances in congenital adrenal hyperplasia cause acne.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

There is no standard treatment for the rare Cronkhite–Canada syndrome, which can be fatal and lead to cancer.

Congenital atrichia with papular lesions is often misdiagnosed, and new diagnostic criteria can improve accuracy.

Early diagnosis and multidisciplinary care are crucial for managing CNOT3 syndrome.

CD271 is crucial for maintaining healthy skin and preventing inflammation.