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A circular RNA helps cashmere goat hair cells become hair follicles by blocking a molecule to boost a gene important for hair growth.

Certain genes are linked to the risk of developing Alopecia Areata.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Nonclassic congenital adrenal hyperplasia is a common genetic disorder that can cause a range of symptoms and requires personalized treatment.

KRT72 gene helps form hair.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

Claudin 6 is crucial for normal skin and hair development.

A new classification for trichothiodystrophy helps identify genetic causes and potential treatments.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Cantú syndrome is caused by mutations in the ABCC9 gene.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The study found two new mutations in a Chinese patient with severe biotinidase deficiency.

A new mouse mutation causes skin and hair issues, influenced by another gene.

A mutation in the human hairless gene causes alopecia universalis.

A gene variant causes a skin and hair disorder by disrupting protein balance.

Lack of TrkC receptor delays hair follicle development.

Defects in certain proteins cause major heart abnormalities during early development.

Lactate production is important for activating hair growth stem cells.

Genetic defects in androgen production are linked to male developmental disorders and are improving treatment understanding.

Researchers identified genes in scalp hair follicles that may affect hair traits and hair loss.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

A specific mutation in the K25 gene causes a rare genetic disorder with curly hair at birth and later hair loss, along with dental issues.

Hair follicle dermal stem cells help control hair growth timing by regulating signals at the hair germ–dermal papilla interface.

FLCN helps control iron levels in cells.

Cyclic ADP-ribose helps regulate calcium and signals that promote hair growth in hair follicle cells.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.