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Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Suppressing ODC activity reduces tumor growth in hair follicles.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Changing the diet of mice lacking the enzyme CBS can affect symptoms related to the genetic condition.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

A new mouse mutation causes skin and hair issues, influenced by another gene.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

The study concluded that hair growth in mice is regulated by a stable interaction between skin cell types, and disrupting this can cause hair loss.

Msx2 deficiency in mice leads to bone growth and organ development problems.

Testosterone significantly affects urination differences between male and female mice.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

The PP2A-B55α protein is essential for brain and skin development in embryos.

The naked mutation in mice causes hair loss and helps identify keratin genes.

A new mouse mutation causes skin and hair defects due to a gene change.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

A mouse gene mutation increases the risk of skin cancer.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Hairless mice lack fur due to a genetic mutation affecting skin response, not hormone issues.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Mice are useful for researching human hair loss and testing treatments, despite some differences between species.