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A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Keratinocytes in hair follicles differentiate similarly to skin cells, with specific patterns in different regions.

pCLCA2 protein may help maintain skin structure and function.

New mutations in the EBP gene cause CDPX2, affecting bones, skin, eyes, and hair, with females generally less affected than males.

A new keratin gene was found in mice, explaining hair growth.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

A specific genetic change in the KRT71 gene causes a hair loss condition in Hereford cattle.

Researchers found a new mutation in the FERMT1 gene in a Spanish family with Kindler syndrome.

The study found nine new hair protein genes in human hair follicles.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Krox20 (Egr2) is important for the function of epithelial stem cells.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

T cell subsets are crucial in kidney cancer, and a new model predicts patient outcomes using key genes.

Two new mutations in the CDH3 gene cause hair loss and vision problems in a young girl.

A cluster of 21 keratin-associated protein genes important for hair growth was found on human chromosome 21.

Mutations in the KRT85 gene cause hair and nail problems.

The man has a genetic skin condition called pachyonychia congenita.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

CK 5/6 expression in breast cancer is linked to negative hormone receptor status and higher tumor grade.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

Certain genetic variants impair enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

NonClassic Congenital Adrenal Hyperplasia is a less severe form of a genetic disorder affecting adrenal gland function.

Deleting the Hoxc13 gene in frogs shows its crucial role in developing skin structures similar to hair.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.