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A new mutation in the ST14 gene causes a rare skin and hair disorder in a specific family.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

HOXC13 is crucial for regulating hair keratin genes in hair follicles.

Keratin 14 may be an autoantigen in autoimmune skin diseases.

K31 can identify clear secretory cells in human sweat glands.

A second domain of high sulfur KAP genes on chromosome 21q23 is crucial for hair structure.

KRT72 gene helps form hair.

Keratin K14 expression varies between hair follicles and epidermis, affecting cell differentiation.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.

Overexpression of Hoxc13 in hair cells causes hair loss and skin issues.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Inward rectifier potassium channels are important in many body functions and diseases, and could be potential drug targets.

Keratin 17 is important for skin's response to radiation, affecting many genes and cell division.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

The study mapped keratin 15 and 19 genes, aiding future genetic disorder research.

CDP is crucial for lung and hair follicle cell development.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

The KAP13-3 gene in sheep affects wool quality by influencing keratin assembly.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

Matriptase is crucial for keeping epithelial tissues healthy and functioning properly.

A specific gene mutation (Y449H in K10) was found in a patient with severe skin disorder.

CLK1 is needed for skin cells to become epidermal cells but not sebocytes.

Topical IKKα inhibitors may help prevent CCS tumours.