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The malignant pilomatricoma showed strong epithelial keratin expression, suggesting it may not calcify.

The gene Foxq1, controlled by Hoxc13, is crucial for hair follicle differentiation.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

Researchers mapped and categorized specific keratin-associated protein genes on human chromosome 21q22.1.

Cathepsin E is crucial for normal skin cell differentiation and development.

A specific gene mutation in KRT86 is linked to hair disorder in a Chinese Han family.

CXXC5 is a protein that prevents hair growth and could be a target for hair loss treatment.

K16 can partially replace K14 but causes hair loss and skin issues.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Connexin-26 gene mutations may increase cancer risk in KID syndrome patients.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Collagen XVII and CD151 affect cell movement, with CD151 inhibiting migration when bound to integrins.

Keratin 75 is important for fast wound healing and works with SOX2 and the LINC complex to help skin cells move and repair damage.

Activated LEF/TCF complexes are crucial for hair development and cycling.

Krtap11-1 is important for hair strength and structure.

Only one of the two K16 genes on chromosome 17 makes a functional protein for keratin filaments.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

Ch55 may help reduce skin scarring and fibrosis.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

A mutation in the KRT25 gene causes woolly hair and hair loss.

A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Krt16-deficient mice help understand skin disorders like PC and FNEPPK.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.