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FOXC1 is essential for keeping hair follicle stem cells inactive and maintaining their environment for healthy hair growth.

Sostdc1 controls the size and number of hair and mammary gland structures.

HDAC6 helps keep ovarian follicles dormant, extending female fertility.

The ITGB6 gene is important for tissue repair and hair growth, and mutations can lead to enamel defects and other health issues.

Scientists found new and known long non-coding RNAs in mouse hair follicle stem cells that may be important for stem cell function and could be targets for cancer treatment.

Dkk4 is necessary for the initial development and arrangement of hair follicles.

Activating β-catenin increases melanocytes and decreases Schwann cells.

CIPK13 and CIPK18 genes are crucial for root hair growth in plants.

FoxN1 overexpression in young mice harms immune cell and skin development.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Pachyonychia congenita is linked to a keratin gene on chromosome 17.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

Antizyme reduces tumor growth and normalizes skin cell development affected by MEK.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

IL-18 signaling helps mature Tregs move into the thymus.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.

Polycomb Repressive Complex 2 is not needed for hair regeneration.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Injecting specific oligonucleotides can change hair growth and structure by altering a gene.

A KRT32 gene variant causes loose anagen hair syndrome.