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A new sheep gene, KRTAP36-1, may help breed sheep with better wool by reducing prickle factor.

Accurate diagnosis and early specialist referral are crucial for managing 46,XY Disorders of Sex Development.

TEDAR is crucial for skin cell differentiation and barrier formation.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Tmem50b and 2610305D13Rik genes play key roles in early mouse embryo development.

New treatments targeting specific genes show promise for treating keratin disorders.

Hirosaki hairless rats have sparse, twisted hair due to missing hair keratin genes.

Different types of cells in the eye express specific keratins at various stages of development.

The malignant pilomatricoma showed strong epithelial keratin expression, suggesting it may not calcify.

Only a few hair-specific keratins are linked to inherited hair disorders.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

cDermo-1 causes dense skin, feathers, and scales in chickens.

KRT80 may worsen cancer by increasing growth and spread, but its full effects on treatment and outcomes need more research.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Scientists discovered a unique hair protein, KAP24.1, with a special structure, found only in the upper part of hair cuticles.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

BMI1 is essential for preventing hair greying and maintaining hair color.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Frontal fibrosing alopecia is linked to four genetic areas, especially the HLA-B*07:02 allele.

Keratin mutations cause skin diseases and could lead to new treatments.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Runx1 and CDK inhibitors help keep hair follicle stem cells inactive, affecting their future roles.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

ROR2 is crucial for hair follicle stem cell maintenance and self-renewal.

Deleting TNFα gene reduces skin cancer risk in certain mice.