Search

everythinglearnresearchcommunity

for

Search:↓

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Certain genetic variants in keratins increase the risk of tooth decay.

CCCA in women of African ancestry may be caused by PADI3 gene mutations and intense hair grooming.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

Two new gene clusters important for hair formation were found on human chromosome 11.

Keratin 75 might be important in oral cancer progression.

All five human steroid 5α-reductase enzymes are found in the endoplasmic reticulum.

Researchers identified a new variant of the FGF5 gene in sheep that affects hair length.

Hair shaft changes may be linked to CCCA, but their role is unclear.

High glycine–tyrosine keratin-associated proteins help make hair strong and maintain its shape.

Keratin K6irs is a marker for the inner root sheath of hair follicles in mice and humans.

Targeting FGF5 could help treat prostate cancer.

CCCA may be a fibroproliferative disorder, and anti-fibrotic therapies could help.

NFIB and STAT5 work together to control specific genetic programs in cells.

Polycomb Repressive Complex 1 is crucial for keeping stem cells stable and maintaining healthy adult tissues.

Pachyonychia congenita is linked to a keratin gene on chromosome 17.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Editing the FGF5 gene in sheep increases wool length, confirming its role in hair growth.

K31 can identify clear secretory cells in human sweat glands.

Loss of keratin K2 causes skin problems and inflammation.

Different tissues in cows have different types of cytokeratins.

A new mouse mutation causes skin and hair defects due to a gene change.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.