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PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

ALKBH5 helps wounds heal faster by stabilizing PELI2 mRNA.

The ovine Hoxc-13 gene is crucial for hair formation and may have other skin functions.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

A new gene location for Keratosis follicularis squamosa was found on chromosome 7p14.3-7p12.1.

IL-6 contributes to hair loss by shortening the growth phase and causing hair follicles to regress.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

The technology can create transgenic cashmere goats with improved wool quality.

Normal cells outcompete and remove mutant cells in the pancreas with the help of the EphA2 receptor.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

BMP6 and Wnt10b control whether hair follicles are resting or growing.

Hair loss in certain mice is linked to changes in keratin-related genes.

Pax9 is crucial for proper tongue surface development and preventing skin-like changes.

The study concludes that certain domains in Clostridium histolyticum enzymes are structurally unique, bind calcium to become more stable, and play distinct roles in breaking down collagen, with potential applications in medicine and drug delivery.

Keratin 17 is important for hair and nail structure and affects pachyonychia congenita symptoms.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Key genes can rewire networks, changing skin appendage types.

MEF2C is crucial for normal hair cycle progression.

Overexpressing CtBP1 in skin cells causes skin and hair problems.

aPKCλ is essential for hair follicle stem cell maintenance and wound healing.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

New mutations in the EBP gene cause CDPX2, affecting bones, skin, eyes, and hair, with females generally less affected than males.

Infants with severe KID syndrome may be more prone to serious infections and need close monitoring.

The study found nine new hair protein genes in human hair follicles.