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Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

Injecting certain cells into mice caused hair loss, which was preventable with a specific inhibitor.

The hairy ear mutation in mice is linked to changes in gene expression affecting hair growth.

Nut extracts promote hair growth in mice.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

Mouse tail skin has different keratinization near hair follicles and scales.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Msx2-deficient mice experience irregular hair growth and loss due to disrupted hair cycle phases.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

The mouse models are effective for testing new hair loss treatments.

β-estradiol slows hair growth in mice.

The spiny mouse can fully regenerate skin after burns, unlike the lab mouse.

Too much zinc in the diet can cause hair loss and color change in young mice by reducing copper in the body.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Merkel cells develop independently of nerves and are linked to specific hair follicles in mice.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

Aire mutation reduces alopecia areata, while Notch4 mutation prevents it in mice.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

The complex oriental medicine extract was as effective as minoxidil for hair regrowth in mice without adverse effects.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

Chamaecyparis obtusa oil promotes hair growth in mice.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.