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Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

The vitamin D receptor can work without its usual activating molecule.

Vitamin D receptor is crucial for normal hair growth and preventing hair loss.

Key genes and pathways influence cashmere production in goats.

VDR gene variations may affect carbohydrate metabolism in young women with hyperandrogenism.

ΔNp63α helps control a protein that stops cancer cells from spreading.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Oral tofacitinib significantly improves lichen planopilaris symptoms without adverse effects.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Vitamin D receptor helps control hair growth genes in skin cells.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Vitamin D receptor is crucial for hair health and may help treat hair loss.