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Different melanocyte types in hair follicles either survive or die during the catagen phase.

The research suggests that the global distribution of PCOS is likely due to historical human migration and that genes affecting PCOS may have different impacts on males and females.

A new model using mice with human hair follicles helps better understand hair loss from chemotherapy.

3D cell cultures produce extracellular vesicles similar to those in the body.

Cysteine-rich keratins evolved independently in mammals, reptiles, and birds for hard skin structures like hair, claws, and feathers.

Targeting TCR-Vβ2 in cutaneous T cell lymphoma shows promise for safer, more specific treatment.

Hair keratins evolved from ancient proteins, diversifying through gene changes, crucial for forming claws and later hair in mammals.

Autoimmune skin diseases result from genetic and environmental factors disrupting immune checkpoints.

The model successfully simulates human hair growth and patterns, including hair loss types.

Cellular flows and tissue mechanics guide feather follicle formation in birds.

Hair in mammals likely evolved from glandular structures, not scales.

Different types of resting melanocyte stem cells have unique characteristics and vary in their potential to become other cells.

The model helps understand alopecia areata and suggests better treatment strategies.

PTCH gene mutations contribute to basal cell carcinoma development.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

scMC effectively separates biological signals from technical noise in single-cell genomics data.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Bcl-2 helps hair regeneration but can also increase cancer risk.

Ancient DNA blocks are still present in human genomes, possibly due to advantages they provide.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

The research shows that skin cancer likely originates from hair follicles and that certain cell populations expand to promote skin cancer growth.

New hair regrowth model introduced, imiquimod kills skin cancer cells, T-cadherin loss makes skin cancer more invasive, no strong link between PTCH1 gene and skin cancer after transplant, and male teens more likely to have hereditary hair loss.

Lineage tracing helps track cell development and has various methods with pros and cons.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

Corneous beta-proteins evolved uniquely in reptiles and birds, forming scales, claws, beaks, and feathers.

Hair growth and shedding involve specific cell changes and gene roles.

The model helps understand alopecia areata and suggests ways to improve treatment by targeting immune issues.

Understanding chaos and control mechanisms in disease can improve diagnosis and prediction in medicine.

Changes in the HR gene have influenced hair growth and may lead to hair loss conditions in humans.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.