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Quiescent cells have increased mitochondrial activity and ECM gene expression, but reduced glycolysis.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Hair follicle cells can become bone-like cells, useful for bone repair.

Scientists created keratinocyte cell lines from human hair that can differentiate similarly to normal skin cells, offering a new way to study skin biology and diseases.

Keratinocytes can reduce the survival of certain melanoma cells, suggesting new therapy paths.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The protocol allows easy imaging of dividing cells in C. elegans and can be used for other organisms.

The flap assay grows the most natural hair but takes the longest, the chamber assay is hard work but gives dense, normal hair, and the patch assay is quick but creates poorly oriented hair with some issues.

Blocking casein kinase 1 in skin cells can help melanocyte precursors move better, potentially helping with conditions like vitiligo or gray hair.

Scientists used a special imaging technique to observe that hair follicle regeneration involves cell division and structural changes, mostly in the lower part of the follicle, and that the dermal papilla at the base is crucial for regrowth.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Lentiviral vector effectiveness in skin is influenced by external factors, not receptor availability.

DNA flow cytometry effectively evaluates how different conditions affect hair growth cell activity.

The new system can grow hair in the lab and test hair growth treatments.

Stat3 activation increases hair follicle progenitors but reduces bulge region stem cells.

Hair cell cloning could potentially rejuvenate shrinking hair follicles, allowing for lifelong hair maintenance.

ALRN-6924 may prevent hair loss caused by chemotherapy.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.

The research found ways to activate melanocyte stem cells for potential treatment of skin depigmentation conditions.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Human skin cells can be turned into heart cells.

Mature cells can re-enter the cell cycle and potentially lead to cancer.

Activin activation in skin cells speeds up wound healing without affecting scar quality.

Stem cells can be used to create long-lasting skin cells for treating pigment disorders.

A new mouse model helps study melanocyte cells using GFP expression.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

Lithium chloride helps hair follicle cells become tooth cells by activating a specific signaling pathway.