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New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

EAAT4 decreases with age, harming skin function and calcium balance.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

A genetic marker linked to a type of hair loss was found in most patients studied.

Mutations in the DSG4 gene cause fragile, sparse hair in humans, mice, and rats.

The research linked PLCD1 gene variants to the development of trichilemmal cysts.

Controlling Tslp can improve health in AEC syndrome patients.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.

PADI4 enzyme slows down cell growth in developing hair follicles.

A girl with excessive hair growth had a genetic change on chromosome 17 that reduced the activity of two genes linked to hair growth.

The trichohyalin gene is located at chromosomal region 1q21 with other skin-related protein genes.

Mutations in the DSG4 gene can cause a rare hair disorder similar to monilethrix.

The enzyme PA-PLA1α is important for proper hair follicle development.

Thymic stromal lymphopoietin (TSLP) promotes hair growth by stimulating specific skin cells.

A gene network led by RSL4 is crucial for early root hair growth in response to cold in Arabidopsis thaliana.

The AR gene is linked to male-pattern baldness, TNFSF4 to heart disease, SLC19A3 to BBGD, MCT8 to a syndrome, and segmental duplications to genetic variation.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The Itpr3 gene causes a specific hair pattern in mice.

TEDAR is crucial for skin cell differentiation and barrier formation.

Truncated LTBP-1 disrupts TGF-β signaling, affecting hair growth.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

LPA 4 helps control blood and lymph vessel development in zebrafish.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

Cathepsin L is essential for heart health and its absence causes heart problems and hair loss.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

HAT-L4 is crucial for preventing body fluid loss by maintaining skin barrier integrity.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

TLR4 is important in aging-related diseases and could be a new treatment target.