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A new skin disease in four Labrador retrievers responded well to immunosuppressive treatment.

IL-17 and certain immune cells are linked to more severe alopecia areata.

Researchers developed promising agents for prostate cancer imaging, with the best one showing high potential for clinical use.

ULBP3 could be a marker for diagnosing alopecia areata incognita and may be linked to its cause and development.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

HAT-L4 is crucial for preventing body fluid loss by maintaining skin barrier integrity.

Cats likely have a reactive skin condition, while dogs may have a more complex, possibly cancerous one.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Feline cutaneous lymphocytosis is likely reactive, while canine cutaneous lymphocytosis needs more study to understand its nature.

Skin-resident memory T cells may contribute to chronic alopecia areata and baricitinib could be a potential treatment.

Tocilizumab therapy may cause skin and hair conditions like halo naevi, vitiligo, and alopecia areata.

PEGylated thymosin β4 shows promise for treating heart attacks effectively.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

BTNL2 helps protect hair follicles from immune attacks.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

IL-4 gene variation may increase the risk of alopecia areata in Turkish people.

Alopecia areata is linked to immune system differences, with specific biomarkers like CXCL9 and CXCL10 being key for diagnosis and potential treatment targets.

LC-OCT is a useful noninvasive tool for diagnosing and monitoring alopecia areata.

All parts of the CLASI-A score are important for assessing skin activity in cutaneous lupus erythematosus.

Alopecia areata is linked to reduced T cell function and auto-immunity.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

People with alopecia areata have higher levels of a heart disease marker in their blood.

Scientists are still unsure what triggers the immune system to attack hair follicles in Alopecia areata.

Cathepsin L deficiency causes large, abnormal cell structures and health issues in mice.

Targeting CXCL10 may help treat alopecia areata.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.