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A woman with X-linked chronic granulomatous disease developed lupus-like skin lesions, improved with treatment, suggesting a unique skin condition in carriers.

A prostate cancer drug can lower the levels of a protein that the coronavirus uses to enter lung cells.

Special gut cells help stem cells move to and fix injured areas by activating a specific signaling pathway.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

The Hedgehog signaling pathway is linked to cancer development, and targeting it with inhibitors shows promise but faces challenges like resistance.

New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Hoxc13 gene is essential for hair, nail, and papilla development.

Increasing calcium sensing receptor speeds up skin and hair development in mice.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

LHX2 and SOX9 identify unique hair follicle cell groups, crucial for hair maintenance.

Early and accurate diagnosis is crucial for managing rare genetic disorders like this localized variant of junctional epidermolysis bullosa.

FCER1A and RGS1 may help diagnose and treat systemic lupus erythematosus.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

Small molecules KY19382 and KY19334 may help treat skin cancer by reducing CDK1 levels and blocking harmful cell signals.

Hox proteins help maintain keratinocyte identity by regulating miRNA expression.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

H19 may help cause PCOS by affecting CTGF levels, suggesting a new treatment target.

Lack of Crif1 in hair follicle stem cells slows down hair growth in mice.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Higher CRBP1 levels are linked to more severe alopecia areata.

OCT4B1 and SOX-2 levels are higher in Crohn's and ulcerative colitis patients, possibly aiding tissue repair.

PTCH gene mutations contribute to basal cell carcinoma development.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

FOXC1 boosts SFRP1 in hair loss, suggesting new treatments.