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Certain herbal compounds might help prevent hair loss in menopausal women by activating Vitamin D receptors.

5α-reductase affects dopamine receptors linked to sensorimotor gating, which may help understand disorders like schizophrenia.

Vitamin D3 and its receptor are important for hair growth, and understanding receptors could help treat hair graying and skin cancer.

Vitamin D and calcium are important for quick and effective skin wound healing.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Vitamin D may help treat hair disorders.

Vitamin D3 affects cell differentiation in specific skin areas.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Vitamin D3 may protect rat hair follicles from radiation damage.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Vitamin D boosts a specific protein in skin cells linked to hair follicles.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Rubbing calcipotriol on the skin works better than taking vitamin D pills for treating mild to moderate alopecia areata.

Vitamin D receptor helps control hair growth genes in skin cells.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.