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As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Low vitamin D levels are linked to various types of hair loss, but more research is needed before using it as a treatment.

Vitamin D might be involved in the development of alopecia areata and could help in its treatment.

Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

A new hyaluronan-based biomatrix successfully supports the growth of mouse ovarian follicles, producing healthy eggs.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Vitamin D is important for skin health, but more research is needed to understand its full effects and treatment potential.

People with certain types of hair loss, especially lichen planopilaris and telogen effluvium, as well as African Americans, Asians, and men, are more likely to have severe vitamin D deficiency.

Alopecia areata patients have lower vitamin D levels than healthy individuals.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor helps control hair growth genes in skin cells.

Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Vitamin D receptor gene variations are not linked to alopecia areata.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Lack of vitamin D receptor causes hair loss in mice by allowing certain genes to overactivate.

Low vitamin D levels are common in people with Alopecia Areata.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D could be important for many skin functions and synthetic versions may treat various skin diseases.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Women losing hair might have lower levels of vitamin D.

Low iron levels are a significant risk factor for hair loss, while high vitamin D levels might be a response to hair loss, not a cause.

Certain variations in the vitamin D receptor gene are linked to an increased risk of polycystic ovary syndrome.

Men and premenopausal women in Korea show different patterns in iron and vitamin D levels, with no clear pattern for postmenopausal women.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

Vitamin D levels are not linked to alopecia areata.