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Introducing the OTC gene improved symptoms in mice with OTC deficiency.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Ifidancitinib, a JAK inhibitor, effectively regrows hair in mice with alopecia by tiring out harmful T cells.

Bim and Puma proteins are found in developing mouse hair follicles and are involved in more than just cell death.

Activating Tgr5 may help treat hair loss and bone loss.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

The mutation helps mice handle heat better without affecting hair growth.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Topical diphencyprone helped regrow hair in mice and rats with a condition similar to human hair loss.

Testosterone significantly affects urination differences between male and female mice.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

Sex steroids affect the MafB gene differently in male and female hamsters.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

A single recessive gene causes sparse hair in certain Japanese White rabbits.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Tranexamic acid turns white hair brown in certain mice by affecting specific proteins.

The DQB1*03 allele is linked to higher alopecia areata risk in Italians.

Parental uveitis increases offspring's risk and severity of autoimmune eye disease.

A new mouse model effectively mimics vitiligo for research and drug testing.

Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

BMD-1141 effectively regrows hair in alopecia areata with less frequent dosing than current treatments.