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Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

DEC cells show promise as a safe and effective treatment for Duchenne muscular dystrophy.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Lhx2 helps retinal cells respond to signals for eye development.

A gene mutation causes lanceolate hair in rats by disrupting hair shaft integrity.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Mice with human chymase had a higher death rate when exposed to a toxin compared to normal mice.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

CTCF protein is essential for skin and hair follicle development in mice.

Type XIX Collagen is present in specific skin and hair cells during development.

The mTurq2-Col4a1 mouse model shows how the basement membrane develops in live mammals.

Turning off the Lhx2 gene in mouse embryos leads to slower wound healing and scars.