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Ent-kaurane-type diterpenoids from Isodonis Herba may promote hair growth by boosting cell proliferation.

A genetic mutation in a specific gene causes a salt-wasting condition in a Pakistani girl and her family.

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

CD2 might be a new treatment target for patchy alopecia areata.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

Inhibiting class I HDACs helps maintain hair growth ability in skin cells.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

Females with 5 alpha-reductase-2 deficiency have less body hair, no acne, normal fertility, and delayed menarche.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

Enzymes that change arginine to citrulline are important for skin barrier and hair formation, and their malfunction can lead to skin conditions and hair disorders.

CD8A and FOXD2-AS1 may be key for diagnosing and treating alopecia areata.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

JAK inhibitors helped treat hair loss in two people with Down syndrome.

Researchers found specific genes in the part of hair follicles that could help treat hair disorders.

INT effectively shows enzyme activity and protein groups in wool and hair follicles.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

The protein DDX6 helps keep skin cells renewing properly by controlling the production of certain other proteins and breaking down those that cause cells to mature too quickly.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

DK peptides can reduce hair loss by inhibiting the 5α-reductase enzyme and have antioxidant effects.

Steroid hormones may affect sexual maturation in cod by altering certain KCa channel expressions.

New genetic variants linked to inherited ichthyoses were identified, offering insights for potential gene therapy.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Over 67 genes linked to ichthyosis help improve diagnosis and treatment.