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TCDD changes skin cell growth and keratin production in mice.

TTD hair brittleness is caused by multiple structural abnormalities.

Human TMEM2 does not break down hyaluronan but helps control its metabolism.

UV exposure damages hair, increasing thiols and altering protein structure.

Mutations in TBX3 cause horses to have more even hair color instead of Dun camouflage.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

Lowering homocyst(e)ine levels can reverse skin and hair lightening by restoring enzyme activity needed for pigmentation.

Tiger tail banding and hair abnormalities are reliable indicators for diagnosing trichothiodystrophy.

A specific gene mutation causes sparse, brittle hair in a family.

Genotyping for NUDT15 p.Arg139Cys can help predict thiopurine side effects in Japanese IBD patients.

TET enzymes are important for skin and hair development by controlling gene activity in specific areas.

Two siblings were diagnosed with Trichothiodystrophy, identified by brittle hair and low sulfur content.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

A new classification for trichothiodystrophy helps identify genetic causes and potential treatments.

The E413K mutation in the hHb6 gene causes monilethrix, a hair disorder, but doesn't show consistent symptoms.

The tested hair dye ingredients do not form harmful oxidized metabolites in the liver.

A gene mutation causes monilethrix in a Chinese family.

Dihydrotestosterone (DHT) doesn't affect rat skin cell growth, but it does change cell cycle, protein levels, and other cell functions, potentially shortening hair growth cycle.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Different harmful mutations in the CDH3 gene cause HJMD, but symptoms vary among individuals.

Human hair medulla doesn't break down hydrogen peroxide, which may affect hair color formation.

Thiopurines help treat IBD but require genetic testing to avoid side effects.

The new antibody, TYHF-1, specifically targets certain hair-related structures.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

Too much thymosin beta4 causes weird teeth and more hair growth in mice.

Personalized thiopurine dosing based on NUDT15 genotyping can improve long-term outcomes for ulcerative colitis and Crohn's disease patients.

The NUDT15 gene variant causes severe side effects from azathioprine in some Indian patients.