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A new sheep gene, KRTAP36-1, may help breed sheep with better wool by reducing prickle factor.

Skin stem cells were turned into heart cells using a chemical, suggesting a new way to treat heart attacks.

Reactive oxygen species and calcium create a feedback loop that shapes root hair cells.

Oxidative dyes damage hair more than semi-permanent dyes, but new technologies help protect hair while coloring.

A gene mutation in mice causes permanent hair loss and skin issues.

KAP6 genes are conserved across species and active in hair follicles.

A new one-step test can quickly identify skin cancer during surgery.

A person got uncombable hair syndrome from two copies of chromosome 1 from their mother.

Stem cell differentiation involves gradual chromatin changes and dynamic gene activity.

Mutations in the SNRPE gene cause hereditary hair loss.

Different versions of the KRTAP6-2 gene in goats can lead to thinner cashmere fibers.

The DNMT3B -579G>T polymorphism may increase the risk of colorectal cancer.

Superoxide dismutase (SOD) can prevent hair graying in mice.

A boy's skin fragility and sparse hair were caused by a genetic mutation affecting skin cell adhesion.

Mutations in three genes cause Uncombable Hair Syndrome, leading to frizzy hair that can't be combed flat.

A girl with excessive hair growth had a genetic change on chromosome 17 that reduced the activity of two genes linked to hair growth.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Dab2 protein is crucial for hair follicle stem cell renewal and preventing early aging.

The new ddPCR method reliably detects unwanted viruses in CAR-T cell products, ensuring their safety for patients.

A TP63 gene mutation causes significant hair loss and mild skin, nail, and tooth abnormalities.

Proteolytic enzymes damaged hair follicle stem cells in transgenic mice.

A rigid compound with a common structural motif was successfully synthesized.

Scientists made two new chemicals that might help treat diseases caused by male hormones.

A KRT32 gene variant causes loose anagen hair syndrome.

Chemical treatments and light exposure damage hair proteins.

Defective nuclear transport may cause gene expression changes in Progeria.

The enzyme system in hair follicles is similar to the liver's and is affected by certain inhibitors.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

The gene URB is more active in human hair growth cells and responds to a hair-related hormone.