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Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

Delta-opioid receptors affect skin cell circadian rhythms, possibly impacting wound healing and cancer.

Both main and alternative Wnt signaling are important for regrowing rodent whisker follicles.

A boy with a new NR5A1 gene mutation has a sex development disorder without affecting his adrenal glands.

Mast cell and nerve fiber interactions in mouse skin change with the hair cycle.

Neutrophils quickly move to the site of skin injury.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Hair follicles and glands significantly increase steroid absorption in skin.

A new EDA gene variant causes X-linked hypohidrotic ectodermal dysplasia in a Chinese family.

Chemical lumbar sympathectomy is an effective and safe treatment for idiopathic livedo reticularis.

A rare gene variant causes sexual development issues in siblings, needing personalized treatment.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

Implanted dermal papillae can induce hair growth in rat ear wounds.

The method allows precise cell removal without harming nearby tissues.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

The model better predicts how water-loving and fat-loving substances move through the skin by including tiny pores and hair follicle paths.

Different species use the same genes for tooth regeneration.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Injecting 6-OHDA in newborn mice delays hair growth and thins skin.

New SRD5A2 variants may disrupt protein function, aiding diagnosis and treatment of related disorders.

Activating TRPV1 can boost hair growth by involving neurons, macrophages, and fibroblasts.

Dynamic, light touch is sensed through a common mechanism involving Piezo2 channels in sensory axons.

The OVOL1 gene, controlled by β-catenin, is crucial for creating hair follicles.

Reducing nerve growth can help skin regenerate after birth.

Two methods improved nerve regeneration and touch recovery in skin grafts for burn patients.