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Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Trichothiodystrophy involves brittle hair due to low sulfur amino acids, not a transport defect.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

A rare gene variant causes sexual development issues in siblings, needing personalized treatment.

Skin cells can naturally limit the growth of cancerous changes by balancing cell renewal and differentiation.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

Increased 14-3-3 proteins may block hair cycle regression, causing hair loss.

Arab APS1 patients have unique and recurrent AIRE gene mutations.

Removing part of the vitamin D receptor stops vitamin D from working properly.

The compound was successfully made and shows potential for treating prostate cancer.

Understanding how EDC genes are regulated can help develop better drugs for skin diseases.

Dexamethasone effectively normalizes elevated steroid levels in girls with premature adrenarche.

Hair transplantation improved hair loss in a coup de sabre patient.

Decorin helps hair cells grow and move, and keeps hair growth phase going in mice.

A girl had rickets due to a gene mutation affecting vitamin D response.

A TP63 gene mutation causes significant hair loss and mild skin, nail, and tooth abnormalities.

Certain cells outside the hair follicle's bulge area can quickly regenerate damaged hair follicles, potentially helping to reduce hair loss from cancer treatments.

Estradiol stops hair growth in mice, but an antagonist can reverse this effect.

CCCA is a scarring hair disorder mainly affecting people of African descent, needing better awareness and treatment.

Anagen effluvium is a reversible condition causing sudden hair loss, often due to chemotherapy or head radiation.

The case suggests that dissecting cellulitis of the scalp can occur in a White child and should be considered when diagnosing pediatric scarring alopecia.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

The CD44-CD49d complex boosts T cell activation and survival in autoimmune disease.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

A new gene variant in the DSP gene is linked to a unique type of hair loss.

Claudin expression changes help the skin respond to injury.