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A new DSG4 gene mutation causes hair defects in a young girl.

Sonic hedgehog signaling is crucial for hair growth and maintaining hair follicle identity.

Sema3A can both slow and speed up wound healing, depending on its form and combination with EGF.

Esrp1 is important for skin health by helping form and maintain the skin barrier.

EBF1 controls hair type and length.

Nelfb is essential for dermal fat development and survival.

The Msi2 protein helps keep hair follicle stem cells inactive, controlling hair growth and regeneration.

ERG increases SOX9, promoting prostate cancer growth and invasion.

SM04755 may be an effective topical treatment for psoriasis.

p63 controls Satb1 to help skin develop properly.

SMAD4 is crucial for muscle repair in young adults but not in aged mice.

Two specific genes are more active during hair growth in mice.

Multiple mouse desmoglein 1 isoforms have distinct roles in skin and hair development.

Pygopus 2 helps ovarian cancer cells grow by aiding ribosomal RNA production, independent of Wnt signaling.

A gene mutation in mice causes skin defects and early death.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

The three estrogen receptor genes are highly expressed in zebrafish neuromasts during development.

Finasteride irreversibly affects human steroid 5α-reductase 2, providing insight into its catalytic mechanism and disease-related mutations.

Human TMEM2 does not break down hyaluronan but helps control its metabolism.

TACE/ADAM17 is essential for maintaining healthy hair and hair follicle stem cells.

Hemidesmosomes and Notch signaling help skin cells mature by moving them to the outer layer.

Activated LEF/TCF complexes are crucial for hair development and cycling.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

DNMT3A is crucial for healthy skin and hair growth.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

BBS7 is crucial for maintaining healthy periodontal ligaments by supporting Shh signaling.

Correcting EDA fibronectin organization and YAP translocation can improve wound healing in fibrotic conditions.

EDA is vital for bone and cartilage formation and could help treat skeletal disorders.